AOR MCP Powder
is on back order
Detoxify and Maintain Healthy Cell Growth
- Contains a soluble source of modified pectin
- Promotes detoxification and chelates heavy metals
- Helps reduce the binding of abnormal cells to healthy cells
- MCP is derived from oranges and is free from masking agents and colors
Supplement Facts Serving Size: 3 scoops Amount Per Serving Modified Citrus Pectin 15 g* *Contains 885 mg of sodium and up to 1350 mg of potassium per day. Non-medicinal ingredients: none.
AOR Guarantees: that no ingredients not listed on the label have been added to the product. Contains no peanuts, eggs, fish, shellfish or any animal byproduct.
Adult Dosage: Take 1 scoop mixed with water or juice 3 times daily with food or immediately after meals (to minimize gastrointestinal irritation and prevent too rapid absorption), or as directed by a qualified health care practitioner.
Cautions: Consult a health care practitioner for use beyond 4 weeks. Diarrhea, stomach irritation, nausea, slowed heart rate and abnormal heart rhythm may occur. Discontinue use and contact your health care practitioner if you develop severe stomach pain, irregular heartbeat or chest pain. Vomiting has been known to occur, in which case discontinue use and consult your health care practitioner.
Pregnancy/Nursing: Do not use
- Metal chelation
- Cellular growth & differentiation
Have you ever heard that the white pith of citrus fruits is good for you? Well it’s true! The white pith just under the rind of citrus fruits like grapefruits and oranges is full of healthy pectin. Unfortunately, the bitter taste and chalky texture makes it quite undesirable to eat. That’s where modified citrus pectin (MCP) comes in. MCP has been shown to be helpful in heavy metal toxicity and in promoting healthy cell growth.
Modified Citrus Pectin as a Heavy Metal Chelator
Heavy metal toxicity is known to be an important factor in declining general health. Adult case reports, one short study in healthy people and one small clinical study in young children have shown tremendous effectiveness for MCP in reducing heavy metal toxicity including lead, mercury, arsenic and cadmium. Larger clinical trials are required for greater conclusiveness, but results like heavy metal reductions by up to 560% are mind-blowing.
Why Citrus Pectin Needs to be Modified
Pectin from citrus fruits contains a significant number of residues of the sugar galactose. This pectin is present in the form of long, complex chains whose galactose residues are hidden deep within their intricate molecular branchings. To solve this problem, the carbohydrate chains can be split into shorter fragments under pH-controlled conditions, exposing more galactose residues and simplifying the complicated branchings of the molecule. Working with the fibers of lemons, grapefruits, or tangerines in this way creates pH-modified citrus pectin (MCP). This modification process maximizes the opportunities for the pectin’s galactose residues to interact with galectin-3, unleashing the potential of citrus pectin.
Lectins & Glycoconjugates: A Lock & Key Mechanism for the Spread of Unhealthy Cells
Galactose is the same kind of sugar residue to which galectin-3 binds. Galectin-3 is a type of lectin. Lectins are cellular communication molecules that allow similar types of cells to bind together (these are called glycoconjugates) and to remain attached to their appropriate tissues. Lectin-glycoconjugate interactions also play a key role in immunity, allowing immune cells to distinguish “self” from “non-self” (foreign) cells, and in other cell-to-cell interactions.
An Unfortunate Match
Its been shown that many unhealthy cells exploit the lectin recognition system by covering themselves with “keys” that match the “locks” in distant, healthy tissues. Either the abnormal cell causes its surface to bristle with a lectin, which then binds to that lectin’s glycoconjugate on a healthy cell type, or vice-versa. Additionally, some unhealthy cells can coat themselves with lectins to allow them to bind to one another, ganging up into clumpings called emboli. This allows abnormal cells that are spreading to more easily escape from your immunological defenses, since the cells on the innermost layers of the emboli may survive even if the immune system destroys cells on the outer layer. By exploiting the match between lectin and glycoconjugate, unhealthy cells are more likely to launch an effective attack.
One lectin “lock” which many abnormal cells learn to “pick” is called galectin-3. Galectin-3 recognizes glycoconjugates, which have the sugar galactose as a key part of its structure (hence gal- (galactose) lectin). By sprouting galectin-3 – or its glycoconjugate – on their surfaces, these cells increase their ability to spread, to take root, and to grow new masses of deadly unhealthy cells. Manipulative abuse of galectin-3 has been implicated in the spread of abnormal cells in the pancreas, colon, lung, ovaries, breast, and especially the prostate. By churning out galectin-3, abnormal prostate cells can easily bind to healthy lung tissue, which is vulnerable to this attack because of its high amount of the lectin’s glycoconjugate; alternatively, by creating more galectin-3 glycoconjugates, abnormal prostate cells find it easier to metastasize to bone tissue.
MCP: A Molecular Decoy Against Metastasis
The idea of using MCP is that the galactose residues on the pectin molecules can provide alternate binding sites for abnormal cells rather than other cells. Animal and clinical studies have certainly found that this is an effective means to reduce the spread of abnormal cells. By delaying abnormal cell growth, MCP provides a larger window of opportunity for other interventions to be more effective.
Other Mechanisms that Promote Normal Cell Growth
MCP can inhibit certain steps of galectin-3-induced angiogenesis (the growth of new blood vessels to feed abnormal cell masses). Galectin-3 can also inhibit abnormal cell anoikis (a defensive mechanism in which abnormal cells are forced to detach from the healthy tissue’s extracellular matrix, leading them to commit cellular “suicide” (apoptosis)), and MCP can block the anti-anoikis action of galectin-3.
Heavy Metal Chelation
Healthy human subjects with no known heavy metal toxicities were given 15g of MCP for 5-days and 20 g on the sixth day. The amount of lead, arsenic and cadmium excreted in the urine was measured each day. After Day 1, arsenic excretion increased by 130%. At Day 6, cadmium excretion had increased by 150%. Over the course, lead excretion had increased by 560%! The chelation is thought to be attributed to the presence of “rhamnogalacturonan II,” which is found in MCP.
In 2007, it was reported that MCP helped reduce lead and mercury, toxic heavy metals, by 74% in five adult patients. This heavy metal reduction was reported to have assisted their return to good health.
Another small pilot clinical study was then published in 2008 in children hospitalized for lead toxicity. Seven children aged 5-12 years were given 15 g of MCP for a month. On average, serum lead levels were reduced by an average of a whopping 161% with no adverse effects.
MCP is known mostly for reducing the spread of abnormal cells. It is also known as a heavy metal chelator.
AOR’s MCP comes from oranges sourced from Florida and from France and not from grapefruits since grapefruit is known to interfere with certain medications. AOR’s MCP also does not contain any masking agents such as colours or flavours in order to maintain a pure product for the vulnerable populations who use this product. This means that the physical properties of the product like colour, taste and solubility are subject to change with environmental factors such as time of harvest, country of origin, soil conditions, etc.